NGF/TrkA-mediated Kidins220/ARMS signaling activated in the allergic airway challenge in mice.

Abstract

These results suggest that Kidins220/ARMS partly participates in the pathogenesis of asthma through the NGF-TrkA signaling pathway, possibly representing a new mechanism in asthma.

Full Text

• DOI - Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology (DOI)
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Subjects

• Animals
• Ankyrins
• Antibodies, Monoclonal
• Asthma
• Bronchi
• Bronchoalveolar Lavage Fluid
• Cells, Cultured
• Disease Models, Animal
• Gene Expression Regulation
• Humans
• Membrane Proteins
• Mice
• Mice, Inbred BALB C
• Nerve Growth Factor
• Receptor, trkA
• Signal Transduction

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Authors

• Xiuqin Ni
• Xing Li
• Xiubin Fang
• Ning Li
• Wanpeng Cui
• Baohui Zhang

Publication date

2010-10-11

Journal

Journal topics

• Asthma
• Hypersensitivity
• Immunity

Language

Eng.

Copyright

Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology

Department of Neurobiology, Institute of Respiratory Diseases, China Medical University, Shenyang, China.

Release reference

Ann Allergy Asthma Immunol. 2010 Oct;105(4):299-306

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