New protein synthesis and trafficking through the Golgi are required for PMA-induced CD36 phosphorylation, suggesting that phosphorylation probably occurs intracellularly.
These studies suggest a novel in vivo pathway for CD36 phosphorylation that modulates cellular affinity for thrombospondin-related proteins to blunt vascular cell signaling.
2012-02-20
Eng.
Arteriosclerosis, thrombosis, and vascular biology
Department of Cell Biology, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, OH, USA.
Arterioscler Thromb Vasc Biol. 2012 Mar;32(3):760-7
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